UCLA professor of neurology and Jeff Bronstein.

UCLA professor of neurology and Jeff Bronstein, UCLA associate professor of neurology Gal Bitan, and report the development of a novel compound as molecular tweezers are known that living model model-synuclein? Blocked form aggregates, held the units of toxicity and further reversed aggregates in the brain that had already formed. And the tweezers achieves this without affecting normal brain function.

The research appears in the current online issue of the journal Neurotherapeutics.There are more than 30 diseases with no cure, protein aggregation protein aggregation and the resulting toxicity to the brain or other organs, including Parkinson ‘s, Alzheimer’s and type 2 diabetes. It is therefore essential, Bronstein said, to find a way to find this aggregation to stop. In the last two decades, researchers and pharmaceutical companies have tried to develop would prevent drugs, abnormal protein aggregation, but so far, they have little or no success.Titles the original article: The adoptive transfer of T-helper cell type 1 clones of attenuates an asthma phenotypes in mice.

-cell clones therapy for bronchial asthma1986 have mouse T helper clone discovered in the two phenotypes, Th1 and Th2 are subdivided.